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給多發性硬化症MS病友和親友的建議:
如要搜尋站內相關文章可多利用
"搜尋此網誌的文章內容"的功能,這樣就可以快速的找到你想要得資訊而不需要從第一篇開始看了.
有關CCSVI(靜脈血管窄化及手術的資訊)可在相關連結以及相關MS blog內

推薦頻道:Gimmy a break

2016年3月28日 星期一

總結:壓力如何讓我們生病

TED上有一擇影片,內容已經說明長期處於壓力之下是如何形成疾病的。

裡面的重點如下:

1. 嚐其分泌壓力荷爾蒙會產生:高血壓,血管內壁細胞無法正常運作,而引發膽固醇累積,動脈硬化-->心臟病,中風
2. 當大腦感到壓力的時候會產生:腸躁症,胃灼熱,腸裡面的細菌改變,降低免疫力,容易感染。
3.壓力會造成:疲倦,注意力不集中,煩躁不安

所以如何應對壓力是現代人的顯學!

好好靜心放鬆吧!




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2016年3月7日 星期一

有沒有可能和壓力成為好朋友呢?

壓力既然是中樞神經發炎的元凶,那有沒有可能和壓力成為好朋友呢?

壓力,讓你心跳加速、呼吸急促跟額頭開始冒汗。上一篇文章說壓力有害於身體健康。但是新的研究調查卻指出,只有當你相信壓力是有害的時候,它才會真正有害於你的健­康。
來看看心理學家Kelly McGonigal的研究:

讓壓力成為你的朋友,視壓力為正面助力:­去接觸身旁的人,關懷他們。

在過去的十年教學生涯中,身為健康心理學家的Kelly教導大家,壓力是自制力的大敵,是影響我們健康的壞東西,最好盡可能讓自己減壓、放鬆。
       

      但在她最新的TED演講中,她承認自己錯了! 原來,只有你把壓力看做是壞東西的時候,壓力才會對你有害若是你能把壓力以平常心看待,並且把你身體因應壓力的變化當作是身體在幫助你接受挑戰。則壓力不但不會影響你的心血管功能,還能激發你尋求外在的社交支持並解決問題。

方法一:重點不是壓力,而是你怎麼面對看待壓力!

有份研究報告花了八年的時間,追蹤美國的三萬名成年人,調查受訪者「過去一年裡你承受了多少的壓力?」 以及 「你是否相信壓力對你的健康是有害的?」 之後他們利用死亡記錄查出哪些人掛了。結果,過去一年裡承受相當大壓力的人,死亡的風險增加了43%,但這只適用於那些相信壓力是有害於健康的人如果承受巨大的壓力的人,並不認為壓力是有害,則他們的死亡風險反而是最低的,而且比那些只承受了一點點壓力的人還低。

 這個研究充份反應出心念重要性:更回應佛家所說的一句話:【萬法由心造】 


 研究統計八年間追蹤的死亡人數,共有18萬2千美國人死於非命,死因非壓力,而是他們相信壓力對身體有害。 這樣算起來每年超過2萬人因此死亡。這樣一來,相信壓力有害身體健康,竟成了美國前15大死亡原因,比凶殺案還多! 

通常在壓力下,我們的心跳速度會加快,呼吸變得急促,開始冒汗,然後,我們會告訴自己不要緊張....  但如果我們能把這個徵兆看作是身體充滿能量,要幫助我們度過挑戰呢? 

方法二:刺激 催產素(oxytocin)賀爾蒙的分泌!

當你與人擁抱時會釋放出來,暱稱【抱抱賀爾蒙】,它會讓你渴望與人有肢體接觸,增強同理心,甚至讓你更願意去幫助你關心的人。而且,催產素是一種隨壓力而生的賀爾蒙,當身體抗壓機制啓動時,你的就會分泌催產素,促使你去尋求幫助,讓你待在關心你的人身邊。更誇張的是,催產素具有純天然的消炎功用,並讓血管在壓力下保持放鬆。

這個研究充份呼應我之前所貼的文章和研究
大家可以參考

擁抱 : 一個強而有力的關懷 2010年8月31日

另類療法外一章:施予,給予2011年12月5日

我最喜歡的一個地方2011年7月29日

 MS 的另類療法:接吻2009年2月11日

我們在生命當中一定多少會面臨主要壓力事件,例如財務困難或家庭風暴,都會讓死亡率上升30%。但是,那些花時間去關心別人的人,壓力對他們的死亡率完全不具任何影響。關心和幫助別人會讓我們從壓力中得到紓解 

這個研究充份呼應了許多句金玉良言:【助人為快樂之本,而快樂的心乃是良藥,施比受更有福。】


總結:
東方人說一命二運三風水,但科學家和數據卻告訴我們,命運不如你的人生的態度重要,而我們的心念和態度決定我們的人生。 

後記:
其實今天看到這樣的研究,有點震驚但也不會太過震驚,畢竟有很多正面和反面的案例在我們的眼前,如何扭轉我們對壓力和挫折的看法和態度才是最困難的地方。也許不是每個人都可以做得到,但是總提供了一個和壓力當好朋友的方法之一。催產素和腎上腺皮質素這兩個荷爾蒙總是平衡著我們人。(難怪我那麼喜歡抱抱....)


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2016年3月6日 星期日

中樞神經發炎的元兇:壓力。


 美國一則報導:
研究指出:壓力大會令人記憶力減退 腦部發炎
http://udn.com/news/story/5/1544652
研究發現,長期承受壓力可能造成記憶力減退,並導致腦部發炎

科學新聞網EurekAlert!報導,美國俄亥俄州立大學研究人員在老鼠身上做實驗,利用較大的老鼠扮演入侵者,對老鼠製造壓力,研究發現,承受壓力的老鼠會記不得迷宮中的逃生出口。
研究發現,老鼠只有在一再被入侵時,才會出現失憶狀況,若只是承受1次壓力,並不會導致記憶力減退。

俄亥俄州立大學副教授高布(Jonathan Godbout)表示,「長期壓力才會導致這種狀況。承受壓力的老鼠想不起逃生出口,但沒有壓力的時候,牠們記得很清楚。」
研究也發現老鼠的腦部出現變化,免疫系統在外部壓力下,導致腦部發炎。研究顯示,短期記憶障礙與腦部炎症及免疫系統有直接關聯。


研究發現,擺脫壓力後,老鼠在28天內可以恢復記憶,但有長達4星期出現迴避社交等抑鬱症狀。
高布表示,研究結果可能應用於醫治長期承受壓力的患者,例如霸凌受害者、軍人,以及被上司欺壓的員工。
研究結果刊登於「神經科學期刊」(Journal of Neuroscience)。

對照之前我在2015年的多篇文章指出

研究人員找到大腦(中樞神經系統) 和免疫系統之間的關聯性了

一個全面性的疾病:MS多發性硬化症 (歸零Reset的人生)

 TED 醫學:睡眠對大腦有多重要-->靜心也是清除大腦廢物的另一種方式

都說明了 壓力和中樞神經的影響極其深遠,有時候如何釋放壓力,如何轉化壓力才是需要正視的。壓力如影隨形。
看看病友們發病的時間點,要嘛就是面臨升學考試,要不就是人生重大變故,要不就是家庭的一些事情,又或者是禍不單行,接踵而來。這些不都是因為承受著自己無法負荷的壓力而讓神經發炎的嗎?


附上原文:
http://www.eurekalert.org/pub_releases/2016-03/osu-lse022916.php

Public Release: 

Long-term stress erodes memory

Study in mice places blame on immune system
Ohio State University

Sustained stress erodes memory, and the immune system plays a key role in the cognitive impairment, according to a new study from researchers at The Ohio State University.

The work in mice could one day lead to treatment for repeated, long-term mental assault such as that sustained by bullying victims, soldiers and those who report to beastly bosses, the researchers say.
"This is chronic stress. It's not just the stress of giving a talk or meeting someone new," said lead researcher Jonathan Godbout, associate professor of neuroscience at Ohio State.
This is the first study of its kind to establish the relationship between short-term memory and prolonged stress. In the case of the mice, that meant repeat visits from a larger, nasty intruder mouse.
Mice that were repeatedly exposed to the aggressive intruder had a hard time recalling where the escape hole was in a maze they'd mastered prior to the stressful period.
"The stressed mice didn't recall it. The mice that weren't stressed, they really remembered it," Godbout said.
They also had measurable changes in their brains, including evidence of inflammation brought on by the immune system's response to the outside pressure. This was associated with the presence of immune cells, called macrophages, in the brain of the stressed mice.
The research team was able to pin the short-term memory loss on the inflammation, and on the immune system.
Their work, which appears in The Journal of Neuroscience, builds on previous research substantiating the connections between chronic stress and lasting anxiety.
The impact on memory and confirmation that the brain inflammation is caused by the immune system are important new discoveries, Godbout said.
"It's possible we could identify targets that we can treat pharmacologically or behaviorally," he said.
It could be that there are ways to interrupt the inflammation, said John Sheridan, who worked on the study and is associate director of Ohio State's Institute for Behavioral Medicine Research.
The mice used in the study are exposed to repeated social defeat - basically dominance by an alpha mouse - that aims to mimic chronic psychosocial stress experienced by humans.
Researchers at Ohio State seek to uncover the secrets behind stress and cognitive and mood problems with a long-range goal of finding ways to help those who are anxious, depressed and suffer from lasting problems, including post-traumatic stress disorder.
This new research focused on the hippocampus, a hub of memory and emotional response.
The researchers found that the stressed mice had trouble with spatial memory that resolved within 28 days. They found that the mice displayed social avoidance, which measures depressive-like behavior, that continued after four weeks of monitoring.
And they were able to measure deficits in the development of new neurons 10 days and 28 days after the prolonged stress ended.
When they gave the mice a chemical that inhibited inflammation, neither the brain-cell problem nor the depressive symptoms went away. But the memory loss and inflammatory macrophages did disappear.
And that led them to conclude that the post-stress memory trouble is directly linked to inflammation - and the immune system - rather than to other damage to the brain. That type of information can pave the way for immune-based treatments, Godbout said.
"Stress releases immune cells from the bone marrow and those cells can traffic to brain areas associated with neuronal activation in response to stress," Sheridan said. "They're being called to the brain, to the center of memory."
###
The researchers' work was supported by the National Institutes of Health, the National Institute of Aging and the National Institute of Dental and Craniofacial Research.
Other Ohio State researchers who worked on the study were Daniel McKim, Anzela Niraula, Andrew Tarr and Eric Wohleb.
CONTACTS: Jonathan Godbout, 614-293-3456; Jonathan.Godbout@osumc.edu John Sheridan, 614-293-3571; John.Sheridan@osumc.edu
Written by: Misti Crane, 614-292-5220; Crane.11@osu.edu

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